Alzheimer ' s: Mobile phones can be responsible for early disease

Alzheimer ' s: Mobile phones can be responsible for early disease

Studies in the last quarter of the century have led to the development of a calcium hypothesis of the origin of Alzheimer's disease. One of the causes of Alzheimer's disease is the excess of intracellular calcium, which is believed to be triggering the mechanisms responsible for the memory loss associated with the disease. Although there have been long-standing doubts about the harmful effects of mobile phones on our body, and in particular on our brain, no study has identified the effects of connected objects as the cause of Alzheimer's disease. Recently, scientists seem to have established a link between early diagnosis in people aged 30 to 40 years and the effects of impulse electromagnetic fields generated by mobile phones. If this conclusion is confirmed, it will mean that the number of Alzheimer's disease may double within 25 years as our generation ages age, which is much more affected by waves.

According to WHO, dementia is the most costly disease in society in the 21st century. The global cost is estimated at $1,300 billion in 2019. By 2030, it is expected to rise to $1,700 billion, or $2,800 billion, given the rising cost of care. 55.2 million people worldwide with Alzheimer ' s disease or related dementia are estimated to increase to 78 million by 2030 and 139 million by 2050 as the population ages. Moreover, almost 10 million new cases are registered each year. That is why the study of the disease, from its causes to its roots, is of paramount importance for the prevention of public health campaigns to the extent possible.

For almost 25 years, researchers have been studying the effects of calcium on the development of Alzheimer's disease, Calthium is acting at the level of our synapses, that is, the electrochemical connections that are formed between our neurons. They rely on the neurotransmitters that form our brain network.

In this context, an innovative study published in the magazine , for the first time, links early cases of Alzheimer ' s disease to the effects of electronic impulse electromagnetic fields, resulting in increased intracellular calcium.

Excessive exposure to EMPs and calcium surplus

Professor Martin L. Pall of the University of Washington, the lead author of the article, has been studying this phenomenon for ten years, and in a press release he explains: "Electromagnetic fields operate through time-changing electrical races and magnetic forces on a nanosecond-scale time scale, with each increase in impulse modularization produced by mobile phones, smart counters, smart cities and self-contained vehicle radars."

Electronic impulse electromagnetic fields. Activation of VGCC results in rapid increases in intracellular calcium. These increases were demonstrated on animal models of Alzheimer ' s disease and revealed the involvement of two pathways leading to Alzheimer ' s disease. Each of the two routes causing pathophysiological effects after EMP exposure is important in the etyology of Alzheimer ' s disease: the route of excessive calcium alarm and the path of peroxinititis/oxidation stress/inflammation.

More and more evidence against our smartphones.

Professor Pal's article summarizes 18 types of evidence linking calcium surplus, excessive wave exposure and early diagnosis of Alzheimer's disease.

Among these evidence, human genetic and pharmacological studies show that high VGCC activity increases the frequency of Alzheimer ' s disease, so it is not only calcium that is important. VGCC activity directly increased by electromagnetic fields also plays an important role in the development of Alzheimer ' s disease.

Second, 12 recent occupational exposure assessments of EMPs have revealed a higher incidence of Alzheimer's disease. Some studies show that EMPs are reducing the normal 25-year latent period of Alzheimer's disease. In addition, longer exposure would exacerbate the disorder because it would have been accompanied by the so-called normal neurodegeneration of our brain. In other words, the longer we use our phones, as is the case with the present generation of 25-30-year-olds, the earlier the disease and more severe symptoms.

The authors subsequently note that in 2013 and 2016 researchers discovered specific changes in the brain of rats exposed to electromagnetic impulses in Alzheimer ' s disease.

However, Professor Pall notes that the Calcium channel blocker VGCC, amlodipine, reduced 11 different brain changes and four behavioural changes in rats, showing the link between calcium levels and its effect on dementia, so it can be an interesting direction in the fight against Alzheimer's disease.

Finally, one of the main points made by Professor Pall is that the age at which Alzheimer's disease began has fallen over the past 20 years, which will coincide with the increased exposure of EMPs to wireless communications, recent studies have reported cases of Alzheimer's disease in the 30-40 age group; in fact, a 2008 report found that two hours of daily exposure to low-level radiation from base mobile phone stations resulted in "massive neurodegeneration" in the brain of young rats. Moreover, a third of rats died within a month.

Therefore, the authors of the study clearly state: "." Simply put, the data show an unusual deterioration in memory, thinking, behaviour and ability to perform daily activities.

Although the number of studies is still low, researchers believe that there is an urgent need to verify the early signs of Alzheimer's disease in people who live near the antenna of small cellular networks for a year or more. Similarly, such tests should be used for persons diagnosed with Alzheimer's disease at an early stage, between 30 and 40 years of age and exposed to electromagnetic fields.

They also recommend analysing Alzheimer's brain markers and doing MRIs to detect anomalies in young people with digital dementia.